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x rat  (Biotium)


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    Biotium x rat
    X Rat, supplied by Biotium, used in various techniques. Bioz Stars score: 95/100, based on 154 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/x rat/product/Biotium
    Average 95 stars, based on 154 article reviews
    x rat - by Bioz Stars, 2026-06
    95/100 stars

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    (A–C) PNN neuroprotective role in mitigating EBRO-induced alterations in levels of apoptotic markers like Bax (A) <t>,</t> <t>Bcl-2</t> (B) , and Caspase-3 (C) in brain homogenates. To identify significant differences between groups, a one-way ANOVA and Tukey’s post hoc test were used for statistical analysis (A–C) . The statistical significance level was set at p < 0.01, and the data were displayed as mean ± standard deviation (SD). There were eight wistar rats (n = 8) in each experimental group. β v/s Sham Control, Vehicle Control, and PNN Perse; δ v/s EBRO; δα1 v/s EBRO + PNN50; δα2 v/s EBRO + PNN100, EBRO + PNN50; and δα3 v/s EBRO + VB12 (30), EBRO + PNN100, EBRO + PNN50.
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    (A–C) PNN neuroprotective role in mitigating EBRO-induced alterations in levels of apoptotic markers like Bax (A) <t>,</t> <t>Bcl-2</t> (B) , and Caspase-3 (C) in brain homogenates. To identify significant differences between groups, a one-way ANOVA and Tukey’s post hoc test were used for statistical analysis (A–C) . The statistical significance level was set at p < 0.01, and the data were displayed as mean ± standard deviation (SD). There were eight wistar rats (n = 8) in each experimental group. β v/s Sham Control, Vehicle Control, and PNN Perse; δ v/s EBRO; δα1 v/s EBRO + PNN50; δα2 v/s EBRO + PNN100, EBRO + PNN50; and δα3 v/s EBRO + VB12 (30), EBRO + PNN100, EBRO + PNN50.
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    (A–C) PNN neuroprotective role in mitigating EBRO-induced alterations in levels of apoptotic markers like Bax (A) <t>,</t> <t>Bcl-2</t> (B) , and Caspase-3 (C) in brain homogenates. To identify significant differences between groups, a one-way ANOVA and Tukey’s post hoc test were used for statistical analysis (A–C) . The statistical significance level was set at p < 0.01, and the data were displayed as mean ± standard deviation (SD). There were eight wistar rats (n = 8) in each experimental group. β v/s Sham Control, Vehicle Control, and PNN Perse; δ v/s EBRO; δα1 v/s EBRO + PNN50; δα2 v/s EBRO + PNN100, EBRO + PNN50; and δα3 v/s EBRO + VB12 (30), EBRO + PNN100, EBRO + PNN50.
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    (A–C) PNN neuroprotective role in mitigating EBRO-induced alterations in levels of apoptotic markers like Bax (A) <t>,</t> <t>Bcl-2</t> (B) , and Caspase-3 (C) in brain homogenates. To identify significant differences between groups, a one-way ANOVA and Tukey’s post hoc test were used for statistical analysis (A–C) . The statistical significance level was set at p < 0.01, and the data were displayed as mean ± standard deviation (SD). There were eight wistar rats (n = 8) in each experimental group. β v/s Sham Control, Vehicle Control, and PNN Perse; δ v/s EBRO; δα1 v/s EBRO + PNN50; δα2 v/s EBRO + PNN100, EBRO + PNN50; and δα3 v/s EBRO + VB12 (30), EBRO + PNN100, EBRO + PNN50.
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    (A–C) PNN neuroprotective role in mitigating EBRO-induced alterations in levels of apoptotic markers like Bax (A) <t>,</t> <t>Bcl-2</t> (B) , and Caspase-3 (C) in brain homogenates. To identify significant differences between groups, a one-way ANOVA and Tukey’s post hoc test were used for statistical analysis (A–C) . The statistical significance level was set at p < 0.01, and the data were displayed as mean ± standard deviation (SD). There were eight wistar rats (n = 8) in each experimental group. β v/s Sham Control, Vehicle Control, and PNN Perse; δ v/s EBRO; δα1 v/s EBRO + PNN50; δα2 v/s EBRO + PNN100, EBRO + PNN50; and δα3 v/s EBRO + VB12 (30), EBRO + PNN100, EBRO + PNN50.
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    (A–C) PNN neuroprotective role in mitigating EBRO-induced alterations in levels of apoptotic markers like Bax (A) <t>,</t> <t>Bcl-2</t> (B) , and Caspase-3 (C) in brain homogenates. To identify significant differences between groups, a one-way ANOVA and Tukey’s post hoc test were used for statistical analysis (A–C) . The statistical significance level was set at p < 0.01, and the data were displayed as mean ± standard deviation (SD). There were eight wistar rats (n = 8) in each experimental group. β v/s Sham Control, Vehicle Control, and PNN Perse; δ v/s EBRO; δα1 v/s EBRO + PNN50; δα2 v/s EBRO + PNN100, EBRO + PNN50; and δα3 v/s EBRO + VB12 (30), EBRO + PNN100, EBRO + PNN50.
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    (A–C) PNN neuroprotective role in mitigating EBRO-induced alterations in levels of apoptotic markers like Bax (A) <t>,</t> <t>Bcl-2</t> (B) , and Caspase-3 (C) in brain homogenates. To identify significant differences between groups, a one-way ANOVA and Tukey’s post hoc test were used for statistical analysis (A–C) . The statistical significance level was set at p < 0.01, and the data were displayed as mean ± standard deviation (SD). There were eight wistar rats (n = 8) in each experimental group. β v/s Sham Control, Vehicle Control, and PNN Perse; δ v/s EBRO; δα1 v/s EBRO + PNN50; δα2 v/s EBRO + PNN100, EBRO + PNN50; and δα3 v/s EBRO + VB12 (30), EBRO + PNN100, EBRO + PNN50.
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    Image Search Results


    (A–C) PNN neuroprotective role in mitigating EBRO-induced alterations in levels of apoptotic markers like Bax (A) , Bcl-2 (B) , and Caspase-3 (C) in brain homogenates. To identify significant differences between groups, a one-way ANOVA and Tukey’s post hoc test were used for statistical analysis (A–C) . The statistical significance level was set at p < 0.01, and the data were displayed as mean ± standard deviation (SD). There were eight wistar rats (n = 8) in each experimental group. β v/s Sham Control, Vehicle Control, and PNN Perse; δ v/s EBRO; δα1 v/s EBRO + PNN50; δα2 v/s EBRO + PNN100, EBRO + PNN50; and δα3 v/s EBRO + VB12 (30), EBRO + PNN100, EBRO + PNN50.

    Journal: Frontiers in Pharmacology

    Article Title: Enhanced therapeutic potential of paeoniflorin and vitamin B12 in intracerebropeduncle ethidium bromide-induced multiple sclerosis-like pathology

    doi: 10.3389/fphar.2026.1792674

    Figure Lengend Snippet: (A–C) PNN neuroprotective role in mitigating EBRO-induced alterations in levels of apoptotic markers like Bax (A) , Bcl-2 (B) , and Caspase-3 (C) in brain homogenates. To identify significant differences between groups, a one-way ANOVA and Tukey’s post hoc test were used for statistical analysis (A–C) . The statistical significance level was set at p < 0.01, and the data were displayed as mean ± standard deviation (SD). There were eight wistar rats (n = 8) in each experimental group. β v/s Sham Control, Vehicle Control, and PNN Perse; δ v/s EBRO; δα1 v/s EBRO + PNN50; δα2 v/s EBRO + PNN100, EBRO + PNN50; and δα3 v/s EBRO + VB12 (30), EBRO + PNN100, EBRO + PNN50.

    Article Snippet: We assessed apoptotic markers, including Bcl-2 [KLR1880; Krishgen Biosystem, Mumbai, India], Bax [KLR0034; Krishgen Biosystem, Mumbai, India], and Caspase-3 [KLR1648; Krishgen Biosystem, Mumbai, India] ( ).

    Techniques: Standard Deviation, Control